TY - JOUR
T1 - Blood compatibility and SOD activity of H2O2-oxidated titanium substrates
AU - Takemoto, S.
AU - Tsuru, K.
AU - Hayakawa, S.
AU - Osaka, A.
AU - Takashima, S.
PY - 2001
Y1 - 2001
N2 - We examined blood compatibility and superoxide dismutase (SOD) activity of titania gels yielded on Ti substrate due to the treatment with H2O2 solutions. They were correlated to the titania gel surface characteristics. Ti subjected to the chemical treatment with H2O2(H2O2-Ti) had high blood compatibility or hardly coagulated, and showed higher SOD activity than Ti without the chemical treatment. Ti treated with a hydrogen peroxide containing TaCl5(H2O2/Ta-Ti) had little lower blood compatibility than H2O2-Ti, and the SOD activity for H2O2/Ta-Ti were lower than that for any other Ti (Ti, H2O2-Ti). Thus the decrease in SOD activity for the plasma contacted with H2O2/Ta-Ti was in part caused by the Ta ions on the titania gel.
AB - We examined blood compatibility and superoxide dismutase (SOD) activity of titania gels yielded on Ti substrate due to the treatment with H2O2 solutions. They were correlated to the titania gel surface characteristics. Ti subjected to the chemical treatment with H2O2(H2O2-Ti) had high blood compatibility or hardly coagulated, and showed higher SOD activity than Ti without the chemical treatment. Ti treated with a hydrogen peroxide containing TaCl5(H2O2/Ta-Ti) had little lower blood compatibility than H2O2-Ti, and the SOD activity for H2O2/Ta-Ti were lower than that for any other Ti (Ti, H2O2-Ti). Thus the decrease in SOD activity for the plasma contacted with H2O2/Ta-Ti was in part caused by the Ta ions on the titania gel.
UR - http://www.scopus.com/inward/record.url?scp=0035155305&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=0035155305&partnerID=8YFLogxK
M3 - Conference article
AN - SCOPUS:0035155305
SN - 1013-9826
VL - 192-195
SP - 35
EP - 38
JO - Key Engineering Materials
JF - Key Engineering Materials
T2 - 13th international Symposium on Ceramics in Medicine (BIOCERAMICS)
Y2 - 22 November 2000 through 26 November 2000
ER -